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We suspect that metabolic/bioenergetic reprograming, observed in this study, could also mediate short-term maintenance of -cell function upon withdrawal of GLP-1 analogues, as documented in humans and rodents by others 21, 33
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doi: 10.1517/13543784.2011.630660 13 GLP-1 receptor agonists in the treatment of type 2 diabetes - state-of-the-art - PubMed
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Peptides 173 , 171149 (2024)
GLP-1 is secreted by L cells in the distal ileum and colon and not only stimulates insulin but also suppresses glucagon, slows gastric emptying, and promotes satiety
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